IglG and IglI of the Francisella pathogenicity island are important

نویسندگان

  • Jeanette E. Bröms
  • Moa Lavander
  • Lena Meyer
  • Anders Sjöstedt
چکیده

4 Jeanette E. Bröms, Moa Lavander, Lena Meyer, and Anders Sjöstedt 5 6 7 Department of Clinical Microbiology, Clinical Bacteriology, and Laboratory for Molecular 8 Infection Medicine Sweden (MIMS), Umeå University, SE-901 85 Umeå, Sweden 9 10 Running title. IglG and IglI impacts F. tularensis virulence. 11 12

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IglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVS.

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Francisella tularensis is an intracellular pathogen that causes the fatal zoonotic disease tularaemia. Critical for its pathogenesis is the ability of the phagocytosed bacteria to escape into the cell cytosol. For this, the bacteria use a non-canonical type VI secretion system (T6SS) encoded on the Francisella pathogenicity island (FPI). Here we show that in F. novicida T6SS assembly initiates ...

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The virulence of Francisella tularensis, the etiological agent of tularemia, relies on an atypical type VI secretion system (T6SS) encoded by a genomic island termed the Francisella Pathogenicity Island (FPI). While the importance of the FPI in F. tularensis virulence is clearly established, the precise role of most of the FPI-encoded proteins remains to be deciphered. In this study, using high...

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تاریخ انتشار 2011